2C-N

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| _other_data=2-(2,5-dimethoxy-4-nitrophenyl)ethan-1-amine

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2C-N, also known as 4-nitro-2,5-dimethoxyphenethylamine, is a psychedelic drug of the phenethylamine and 2C families.[1] It is taken orally.[1]

2C-N was first synthesized by Alexander Shulgin and was described in his 1991 book PiHKAL (Phenethylamines I Have Known and Loved).[1]

Use and effects

In his book PiHKAL (Phenethylamines I Have Known and Loved), Alexander Shulgin lists 2C-N's dose range as 100 to 150Script error: No such module "String".mg or more orally and a duration of 4 to 6Script error: No such module "String".hours.[1][2] It has an estimated typical dose of about 120Script error: No such module "String".mg orally.[2] Its onset is within 30Script error: No such module "String".minutes and peak effects occur after 1Script error: No such module "String".hour.[1] The effects of 2C-N have been reported to include some visual changes, similarities to MDMA, lightheadedness, eye wiggling, easier conversation, and improved mood.[1] It was described as a "strange material, but okay".[1]

Interactions

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2C drugs like 2C-N are known to be metabolized by the monoamine oxidase (MAO) enzymes MAO-A and MAO-B.[3][4] Monoamine oxidase inhibitors (MAOIs) such as phenelzine, tranylcypromine, moclobemide, and selegiline may potentiate the effects of 2C drugs like 2C-N.[3][4][5] This may result in overdose and serious toxicity.[5][3]

Pharmacology

Pharmacodynamics

2C-N activities
Target Affinity (Ki, nM)
5-HT1A 1,450–2,200
5-HT1B >10,000
5-HT1D 832
5-HT1E 676
5-HT1F ND
5-HT2A 23.5–72.4 (Ki)
32.2–170 (EC50Tooltip half-maximal effective concentration)
48–100% (EmaxTooltip maximal efficacy)
5-HT2B 123 (Ki)
730 (EC50)
74% (Emax)
5-HT2C 162–370 (Ki)
ND (EC50)
ND (Emax)
5-HT3 >10,000
5-HT4 ND
5-HT5A >10,000
5-HT6 251
5-HT7 >10,000
α1A >15,000
α1B, α1D >10,000
α2A 240–1,300
α2B 2,240
α2C 891
β1β3 >10,000
D1 19,000
D2 6,100–>10,000
D3 20,000
D4, D5 >10,000
H1 >25,000
H2 >10,000
H3 5,500
H4 >10,000
M1M5 >10,000
I1 ND
σ1, σ2 >10,000
ORs >10,000
TAAR1Tooltip Trace amine-associated receptor 1 >20,000 (Ki) (mouse)
340 (Ki) (rat)
15,000 (EC50) (mouse)
250 (EC50) (rat)
>10,000 (EC50) (human)
28% (Emax) (mouse)
59% (Emax) (rat)
SERTTooltip Serotonin transporter 32,000 (Ki)
154,000 (IC50Tooltip half-maximal inhibitory concentration)
ND (EC50)
NETTooltip Norepinephrine transporter >30,000 (Ki)
287,000 (IC50)
ND (EC50)
DATTooltip Dopamine transporter >30,000 (Ki)
>900,000 (IC50)
ND (EC50)
MAO-ATooltip Monoamine oxidase A ND (IC50)
MAO-BTooltip Monoamine oxidase B 66,000 (IC50)
Notes: The smaller the value, the more avidly the drug binds to the site. All proteins are human unless otherwise specified. Refs: [6][7][8][9][10][11][12]

2C-N is a low-potency partial agonist of the serotonin 5-HT2A, 5-HT2B, and 5-HT2C receptors.[7][9][13][11]

Chemistry

Properties

Salts of 2C-N have a bright yellow to orange color due to the presence of the nitro group,Script error: No such module "Unsubst". unlike all other members of the 2C family in which the salts are white.

Synthesis

The chemical synthesis of 2C-N has been described.[1] It is synthesized by the mixed acid nitration of 2C-H using sulfuric acid and nitric acid.[1]

Analogues

Analogues of 2C-N include DON, 2C-CN, and 25N-NBOMe, among others.

History

2C-N was first described in the scientific literature by at least 1991.[1]

Society and culture

Legal status

Canada

As of October 31, 2016, 2C-N is a controlled substance (Schedule III) in Canada.[14]

United Kingdom

2C-N and most (possibly all) other compounds featured in PiHKAL are illegal drugs in the United Kingdom.

United States

In the United States, 2C-N is a Schedule 1 controlled substance.[15]

See also

References

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  1. a b c d e f g h i j k Script error: No such module "citation/CS1". 2C-N Entry in PiHKAL
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External links

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